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The chances are one half. You will most likley have 2 children without the gene, and 2 with one gene. Since the normal parent has the dominate allele, the Cystic Fiborisis will not be present.

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Q: What are the possible phenotypes of the offspring between a parent who is a heterozygous carrier for cystic fibrosis and a homozygous normal parent?
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What is the probability of a child having cystic fibrosis if one of her parents was homozygous dominant and the other was a carrier for cystic fibrosis?

The probability of a child having cystic fibrosis in this scenario is 50%. If one parent is homozygous dominant (no cystic fibrosis allele) and the other is a carrier (heterozygous), there is a 50% chance of passing on the cystic fibrosis allele to their child, resulting in the child having cystic fibrosis.


What does heterozygous have to do with cystic fibrosis?

Individuals with cystic fibrosis have a mutation in the CFTR gene. Being heterozygous for the mutant CFTR gene means having one normal and one mutant copy of the gene, resulting in a carrier state. Carriers are typically unaffected by cystic fibrosis but can pass the mutant gene on to their offspring.


Is incomplete dominance a characteristic of cystic fibrosis?

No, cystic fibrosis is not an example of incomplete dominance. Cystic fibrosis is an autosomal recessive genetic disorder caused by mutations in the CFTR gene. Incomplete dominance is a genetic trait where the heterozygous phenotype is intermediate between the two homozygous phenotypes.


Why does a person with Cystic Fibrosis have the genotype cc?

Cystic Fibrosis is inherited as a recessive trait. This means that both parents have to carry the allele for the defect. Both parents are Cc which means that 25% of their children will be CC (homozyous dominant and will not be able to pass the recessive allele in subsequent generations, 50% will be Cc(carriers) and 25% will be cc (homozygous reccessive and have Cystic Fibrosis).


What is the medical term meaning liver inflammation and fibrosis?

The medical term for liver inflammation and fibrosis is "hepatitis." This condition can lead to scarring of the liver, known as fibrosis, and if left untreated, can progress to cirrhosis, where the liver becomes severely scarred and may eventually fail.

Related questions

An individual heterozygous for cystic fibrosis?

Is a carrier of cystic fibrosis


How do you deterime phenotypes?

phenotypes are decided by the alleles for that particular characteristic, by a dominant or two recessive alleles. For example, cystic fibrosis has a recessive allele so the phenotype of cystic fibrosis would only appear if there were two of the recessive allele, one from each parent, were present. A heterozygous carrier of the cystic fibrosis allele would show the phenotype of not having cystic fibrosis. So to determine the phenotype simply find out which allele is dominant and find what alleles each parent has the the probability of each phenotype can be calculated


What is the probability of a child having cystic fibrosis if one of her parents was homozygous dominant and the other was a carrier for cystic fibrosis?

The probability of a child having cystic fibrosis in this scenario is 50%. If one parent is homozygous dominant (no cystic fibrosis allele) and the other is a carrier (heterozygous), there is a 50% chance of passing on the cystic fibrosis allele to their child, resulting in the child having cystic fibrosis.


Is cystic fibrosis carried on a dominant of recessive gene?

Cystic fibrosis is an autosomal recessive genetic disease.


What does heterozygous have to do with cystic fibrosis?

Individuals with cystic fibrosis have a mutation in the CFTR gene. Being heterozygous for the mutant CFTR gene means having one normal and one mutant copy of the gene, resulting in a carrier state. Carriers are typically unaffected by cystic fibrosis but can pass the mutant gene on to their offspring.


A example of a homosygous genetic disorder?

Cystic Fibrosis is a recessive homozygous genetic disease


How do you got cystic fibrosis?

Cystic Fibrosis is a genetic disorder, 2 parents have a CF gene, if their offspring inherits that trait they will have Cystic Fibrosis.


What letters will you have if you are homozygous for cystic fibrosis?

i dont know eat a potato OH, and how did you remember how to spell that


Is incomplete dominance a characteristic of cystic fibrosis?

No, cystic fibrosis is not an example of incomplete dominance. Cystic fibrosis is an autosomal recessive genetic disorder caused by mutations in the CFTR gene. Incomplete dominance is a genetic trait where the heterozygous phenotype is intermediate between the two homozygous phenotypes.


Why can Cystic fibrosis be inherited even if neither parent has the disease?

Cystic Fibrosis is an example of a disease inherited as an autosomal recessive genetic disorder. Both parents of a baby who has Cystic Fibrosis have one abnormal recessive gene and one normal dominant gene and are therefore, Cystic Fibrosis carriers. Carriers are said to be heterozygous because they contain one normal copy of the gene and one abnormal copy. A baby who inherits cystic fibrosis has inherited the abnormal gene from both parents and is therefore said to be homozygous recessive.


If both parents have cystic fibrosis what is the probability that one child will have it?

It depends weather or not the woman carries the cystic fibrosis alele. If she carries a recessive CF allele, then the chances of her and the man having a child with CF is 1/4 But if she doesn't carry it then the child wont have CF but could carry the disorder as a recessive allele. again, the chances of that are 1/4.


What is the genotype of two normal parents who have a child with cystic fibrosis?

The genotype would have to be homozygous recessive if the child was completely effected by the disease. The "normal" paretns would have to have heterozygous recessive genotypes. This makes sense since the allele that causes sickle cell shows incomplete dominance when present with a normal allele in a pair. The "normal" parents actually would have a mixture of sickle cell shaped red blood cells combined with normal shaped ones. The carrier parents does not display symptoms of the disease since the regular red blood cells alone can fill the body's need for oxygen under normal circumstances. The only time the cArrier would notice would be under times of extreme oxygen demand, such as a sprint.