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baby fails to cry,an attempt is made to breathe,but there is primary gasps and subsequent raise in heart beat and blood pressure.after one minute there is primary apnoea.it there is no resuscitation,there will be secondary gasps,a flat heart beat and a rapid drop in blood pressure and in five to eight minutes slight brain damage begins.there will be long gasps and long apnoeas and the baby will be acidotic due to low levels of ph in the blood,hypoxia and hypercapnoea.brain damage becomes more severe,there is cerebral oedema and rupture of small blood vessels resulting in cerebral haemorrhage.there is ischaemia of brain cells resulting in severe brain damage.signs are convulsions,inability to suck,baby is more flaccid,cerebral palsy.death then follows as a result of failure to resuscitate.

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12y ago
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Q: What is the pathophysiology of asphyxia neonatorum?
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