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Nolvadex is a type of medicine originally intended to prevent and treat Breast cancer. Since breast tumors are fed through estrogen, Nolvadex works by binding first with the receptor cells before estrogen can make it to these tissues, thus preventing tumor growth. Apart from this, Nolvadex is also used for men with gynecomastia, especially those who are into bodybuilding. Nolvadex is used as a post-supplement to prevent breast enlargement in males.
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What is the generic name for nolvadex?
tamoxifen
What does Tamoxifen look like?
Tamoxifen is available with a prescription under the brand name Nolvadex. Other brand or generic formulations may also be available. Ask your pharmacist any questions you have about this medication, especially if it is new to you. * Nolvadex 10 mg--round, biconvex, white, uncoated tablets * Nolvadex 20 mg--round, biconvex, white, uncoated tablets * Soltamax 10 mg/5ml oral solution
What is the diet supplement havoc?
It is a Prohormone. It is basically a steroid taken orally and is not as powerful as an injectable steroid. Still requires a proper diet, workout regime, cycle support products as also post cycle support (including Nolvadex, clomid, or toremefine)... Hope this helps get you started...
Is it illegal to order nolvadex online?
By using a search engine you can find all kinds of conflicting information and opinion on this drug and its uses. Some jurisdictions appear to classify it as a prescription drug (which would be illegal to possess without a valid prescription). You will have to do your own reserch - form your own opinion - and come to your own conclusion.
How does tamoxifen?
Tamoxifen, or commonly for its brand name as Nolvadex, works by binding into the breast tissues before estrogen can bind on the tissue receptors. As what we might know, Breast cancer is fed through estrogen. However, tamoxifen does not suppress estrogen production, which is one of its advantages. Therefore, other parts of the body can still benefit with estrogen
Does stanazol raise estrogen levels?
You're talking about Winstrol (stanozolol) right? It's mainly used for muscle hardening and fat-burning, it's not capable of converting into estrogen, so no, it doesn't. You also don't have to worry about gynecomastia with it. If you stack with testosterone though, you'll need an anti-estrogen such as Clomid or Nolvadex. Make sure you know what you're doing before you take that crap though.
How does tamoxifen work?
Most breast cancers are Estrogen fueled , called receptor positive. After treatment, most women with that type of breast cancer are put on Tamoxifen for 3-5 years , it is an Estrogen blocking drug and can aid in preventing the BC from returning.
What are the withdrawal effects of tamoxifen?
"The known, serious side effects of tamoxifen are blood clots, strokes, uterine cancer, and cataracts. Other side effects of tamoxifen are similar to the symptoms of menopause. The most common side effects are hot flashes and vaginal discharge. Some women experience irregular menstrual periods, headaches, fatigue, nausea and/or vomiting, vaginal dryness or itching, irritation of the skin around the vagina, and skin rash. As with menopause, not all women who take tamoxifen have these symptoms. Men who take tamoxifen may experience headaches, nausea and/or vomiting, skin rash, impotence, or a decrease in sexual interest."Source:http://www.cancer.gov/cancertopics/factsheet/Therapy/tamoxifenThe anticancer drug tamoxifen (Nolvadex) increases the risk of cancer of the uterus in some women. It also causes cataracts and other eye problems.
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Is there medicine to prevent breast cancer?
Modern western medicine teaches that there is no herbal remedy for breast cancer. As with other cancers, a breast cancer diagnosis can have a dramatic psychological impact on the patient, and it's not at all uncommon for patients in panic to look for a panacea -- a complete and total "easy" fix for the malignancy. And so many turn to herbal remedies, as the course of "treatment" offered is a lot more attractive than the standard course for breast cancer today. This decision can be damaging, in that it often delays prompt and consistent treatment for the malignancy, sometimes past the point of recovery. Systemic herbal remedies are therefore considered dangerous, and the recomendation is to avoid them However, there are herbal remedies that can be applied to minimze the effects of the more conventional treatments. Chemotherapy, for instance, can damage the nail beds -- and for this, Tea Tree Oil applied topically really performs very well indeed. Diet also plays a major role in the treatment regimine for breast cancer. If you're concerned about this disorder, my very best advice is to spend a LOT of effort in selecting a really excellent oncologist, and then consulting with them in great depth. Today's really top notch oncologists do no rule out herbal solutions, but you Do need to really know when and when not to apply them.
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List of all drugs which cross blood brain barrier?
Drug ClassGeneric Name(Trade Name)AbsorptionDistributionElimination Alkylating Agentsmelphalon(Alkeran)oral - variably and incompletely absorbed from the GI tract, decreased in presence of foodmoderately high protein binding, Vz 0.5 l/kgdeactivated in plasma by hydrolysiscyclophosphamide(Cytoxan)oral - high bioavailability; intravenouscrosses blood-brain barrier (limited)hepatic biotransformation (includes activation); 5 - 25% eliminated unchanged (renal); parent and metabolites eliminated in urine are toxic to bladderifosfamide(Ifex)intravenous infusion onlyactive metabolites cross blood-brain barrier (limited)hepatic biotransformation (includes activation); 10 - 60% eliminated unchanged (increases with increasing dose - renal); parent and metabolites eliminated in urine are toxic to bladderbusolfan(Myleran)oral - completely absorbed from GI tractrapid hepatic biotransformationprocarbazine(Matulane)oral - Rapidly and completely absorbed from the gastrointestinal tract.Crosses blood-brain barrier.Hepatic biotransformation, very short elimination half-life. 70% renal elimination as metabolites.dacarbazine(Otic-Dome)Intravenous onlylimited access to CNS; low protein bindingExtensive hepatic biotransformation; 50% renal elimination (1/2 unchanged).Antibioticsdaunorubicin(Cerubidine)intravenous onlybody water, excluded by blood-brain barrierhepatic metabolism produces both active and inactive metabolites.daunorubicin or doxorubicin liposomes(Daunoxome or Doxil)intravenous infusion onlylimited to vascular fluid, animal studies indicate delivery to CNS; tissues selectively "acquire" liposomes.greatly reduced hepatic metabolism compared to un-encapsulated drugdoxorubicin(Adriamycin)intravenous onlyhigh protein binding; extensive uptake into many tissues, does not cross blood-brain barrierhepatic metabolism produces both active and inactive metabolites; tissue metabolism results in production of free radicals.idarubicin(Idamycin)intravenous infusion onlyextensive tissue binding of both native drug and metabolite; very high plasma protein bindinghepatic and extrahepatic metabolism to equipotent metabolite; elimination primarily biliary as active metabolite.plicamycin(Mithracin)intravenous infusion onlycrosses blood-brain barrier, concentrated in Kupffer cells, renal tubular cells and bone surfaceselimination is renalmitomycin (Mutamycin)intravenous onlydoes not cross blood brain barrierhepatic biotransformation, 10% eliminated in urine unchanged (% increases as dose increases)pentostatin(Nipent)intravenous onlycrosses blood-brain barrier (CSF concentrations ~10% of plasma concentrations within 24 hours). Low plasma protein bindinghepatic biotransformation, 30% - 70% eliminated in urine as unchanged drugmitoxantrone(Novantrone)intravenous infusion onlyrapid extensive distribution to tissues; high protein bindinghepatic; long half-life (due to tissue binding & slow metabolism); small fraction eliminated unchanged.dactinomycin(Cosmegen)intravenous onlydoes not cross blood brain barrierMinimal biotransformation; Elimination primarily biliary/fecal 50% unchanged (24 hours), another 10% unchanged in urine (24 hour); remainder of the drug is recovered within 1 week.Antimetabolitesfluorouracil(Adrucil)intravenous onlygood tissue penetration, crosses blood brain barrierhepatic metabolism produces 2 active metabolites and catabolism; respiratory elimination as carbon dioxide; 7 - 20% unchanged in urinecapecitabine(Xeloda)oral (pro drug)as for fluorouracilhepatic activation by conversion to 5 fluorouracil; elimination pattern as for 5 flurouracilfludarabine(Fludara)intravenous onlydistributed to whole body waterRAPIDLY dephosphylated in serum to 2-fluoro-Ara-A, then phosphorylated intracellularly to active compound. Elimination is renal, approximately 20% unchanged 2-fluoro-Ara-Amercaptopurine(Purinethol)oral - variably and incompletely absorbed from the GI tract (up to 50%)Crosses blood-brain barrier but poorly; low protein bindingHepatic metabolism for both activation and catabolism; degraded by xanthine oxidase; 7 - 40% eliminated unchanged.gemcitabine(Gemzar)intravenous infusion onlydistribution of active metabolite is limited by saturable process. Giving gemcitabine at an excess rate WASTES drug (eliminated intact before conversion).Intracellular metabolism (saturable) to active metabolites. Hepatic deamination to inactive uracil metabolite.Hormonal Oncologicstopotecan(Hycamtin)intravenous infusion onlygood tissue penetration, volumes approximately 2x body water, crosses blood brain barrierreversible pH-dependent hydrolysis to inactive moeity (low pH favors active compound), hepatic metabolism insignificant; 30% eliminated unchanged in urineleuprolide(Lupron)IM injection - 90% bioavailability; 1 month, 3 month and 4 month release formulationsdistributed to extracellular fluid volume; moderate (50%) protein binding.Metabolized to several inactive peptides. Less than 5% recovered as parent or pentapeptide metabolite.tamoxifen(Nolvadex)oral administration, bioavailability??Hepatic biotransformation with enterohepatic circulation. Prolonged elimination; Elimination primarily biliary/fecal, mostly as metabolitesMitosis inhibitorsetopside(VP16)oral - variable dose-dependent oral bioavailability (F decreases as dose increases); intravenousLow and variable into CSF, concentration differentials between normal and cancerous tissues. Very high protein binding (97%). Protein displacement interactions and hypoalbuminemia are concerns.Hepatic biotransformation; up to 50 - 60% renal elimination (2/3 as unchanged drug); remainder fecal.Othershydroxyurea(Hydrea)Well absorbed following oral administration.Crosses the blood-brain barrier (very small molecular weight).Hepatic metabolism (inactivation), 80% renal elimination within 12 hours (50% unchanged); balance eliminated from lungs as CO2paclitaxel(Taxol)intravenous onlyextensive extravascular distribution and/or tissue binding. Very high plasma protein binding.Hepatic p450 metabolism. Elimination primarily biliary / fecal. Variable renal elimination of unchanged drug.docetaxel(Taxotere)intravenous onlywidely distributed in tissues; slightly larger than body water; poor CNS penetration.Hepatic p450 metabolism. Elimination primarily biliary / fecal.cisplatin(Platinol)intravenous onlydoes not penetrate CNSrapid non-enzymatic conversion to inactive metabolites. Elimination usually expressed as recovered platinum (only 50% after 5 days), platinum detected in tissues for months.aspariginase(Elspar)intravenousintramuscularslow sequestration by reticuloendothelial system; poor CNS penetrationunknown pathway, only trace amounts appear in the urine following IV administration.Anti-toxicityamifostine(Ethyol)intravenous infusion onlywide rapid distributionmetabolised by alkaline phosphatase to active free thiol metabolite (binds cisplatin metabolites and alkylating agents and scavanges free radicals. Reaction favored in normal tissues (higher AP)dexrazoxane(Zinecard)intravenous onlydistributed to whole body water, low protein bindingseveral hepatic metabolites, intracellular metabolite may be responsible for action though this is speculative at this timemesna(Mesnex)intravenous onlyvolume of distribution approximates body water.rapid hepatic biotransformation to mesna disulfide; mesna disulfide is reduced to mesna by renal tubular epithelium, mesna binds and detoxifies metabolites of oxazophosphorines.I think it maybe useful for You :)
