smooth muscle
vasoconstriction
Vasodilation in the skin and viscera results from the relaxation of smooth muscle cells in blood vessels. This relaxation causes the blood vessels to widen, increasing blood flow to the skin and internal organs. Vasodilation helps regulate body temperature and can occur in response to various stimuli like heat, exercise, or injury.
Peristalsis
stress-relaxation responsestress-relaxation response
Increased sympathetic tone leads to the release of norepinephrine, which binds to alpha-1 adrenergic receptors on arteriolar smooth muscle. This binding initiates a signaling cascade that ultimately results in the relaxation of the smooth muscle, causing the arterioles to dilate.
Vasodilation
norepinephrine. Acetylcholine mainly causes smooth muscle contraction, while norepinephrine can either cause contraction or relaxation depending on the type of receptor it binds to on the smooth muscle cell.
A musculotropic substance acts on muscle tissue. It specifically targets and affects smooth muscle cells, resulting in changes to muscle contraction and relaxation.
The contraction and relaxation of the smooth muscle in their walls can change their diameter, thus changing the level of pressure (large diameter yields low pressure and smaller diameter yields high pressure).
Smooth muscle is controlled by the autonomic nervous system, which includes both sympathetic and parasympathetic divisions. The sympathetic nervous system typically causes smooth muscle contraction, while the parasympathetic nervous system usually promotes relaxation of smooth muscle. Additionally, hormones and local factors can also influence the contraction and relaxation of smooth muscle.
The sarcoplasmic reticulum is a special type of smooth endoplasmic reticulum. It releases calcium ions during muscle contraction and absorbs them during relaxation.