In pulmonary arteries, PO2 is around 40 mmHg and PCO2 is around 46 mmHg. In pulmonary veins, PO2 is around 100 mmHg and PCO2 is around 40 mmHg. In systemic arteries, PO2 is around 100 mmHg and PCO2 is around 40 mmHg. In systemic veins, PO2 is around 40 mmHg and PCO2 is around 46 mmHg.
blood entering the lungs has a partial pressure of oxygen (PO2) of 40 mmHg and a partial pressure of carbon dioxide (PCO2) of 46 mmHg; alveoli, on the other hand, have a PO2 of 105 mmHg and a PCO2 of 40 mmHg. As the blood moves past the alveoli, oxygen and carbon dioxide will diffuse down their respective partial pressure gradients. Oxygen will move from the alveolar space (PO2 of 105 mmHg) to the blood stream (PO2 of 40 mmHg). Carbon dioxide will move from the blood (PCO2 of 46 mmHg) to the alveolar space (PCO2 of 40 mmHg). As the blood leaves the alveolus, the PO2 and PCO2 will have essentially equilibrated with the alveolar air.
Teflon is used for the membrane of pco2 electrodes as it allows for the diffusion of co2 but not ions.
PvO2 = 40mm Hg, PvCO2 = 46mmHg
Metabolic acidosis
In healthy subjects the mean PCO2 fall 18 mm Hg from the baseline and mean PO2 rise 7 mmHg
No. Once blood reaches the alveoli it will immediately start to release CO2 to the air in our lungs and absorb O2 from the air in our lungs. This means that the PCO2 will be higher in the blood of the pulmonary artery than in the blood of the alveolus. If this were not so, then passing blood through the alveoli would be pointless.
Arterial po2 will not change because it's almost at maximum already. Venous po2 will decrease due to increased oxygen consumption by respiring muscle. Venous and arterial pCo2 will actually either stay the same or fall due to the increased ventilation stimulated by the increased Co2 production by respiring muscles. The increased pCO2 is detected by central and peripheral chemoreceptors and leads to increased ventilation, resulting in increased ventilation - causing pCo2 to remain normal or decrease. This mechanism cannot be used to explain the ventilation increase in light exercise because pCo2 hardly rises at all during light exercise, therefore the chemoreceptors may not be responsible for the mechanism resulting in increased ventilation,
"Inspired air " is the stuff around us, so O2 is about 20% & CO2 is about .003 % ... and why? is because of plants and chlorophyll.
No, firstly pO2 is not a particularly good term for the measurement of oxygen within the blood as most of it is tied up in the heamoglobin molecules and as such is not part od the pO2. Secondly the pulmonary artery is the artery that carried deoxygenated blood from the heart to the lungs where they gain oxygen from the alveolar cavity. Under the laws of diffusion this means the pO2 in the alveoli must be higher than the "pO2" in the blood here, but even just common sense tells you that the oxygen levels in the blood here are very low as this is the whole point in the blood going to the lung.
Although venous levels change, arterial Pco2 and Po2 levels remain surprisingly constant during exercise. In fact, Pco2 may even decline to below normal and Po2 may rise slightly because of the efficiency of the respiratory adjustments. Increased blood flow does not cause a change in gas pressures. the only way to change gas pressures is by altering atmospheric pressure, ie. scuba diving, or changing elevation. Partial pressure of any blood born gas is always directly proportional to atmospheric pressure as evidenced by Dalton's law of partial pressures. However in exercising muscle metabolic processes temporarily increase Pco2 and decrease Po2 until equalized by sufficiently oxygenated arterial blood. So the short answer to your question is that intramuscular Pco2 pressures would increase and Po2 would decrease, but the partial pressures in the blood would remain constant.
The highest partial pressure of O2 (PO2) in the body typically occurs in the alveoli of the lungs, where oxygen is exchanged between air and blood. This ensures efficient oxygen uptake into the bloodstream for delivery to tissues.