Ach (acetylcholine) is a neurotransmitter that is released by motor neurons at the neuromuscular junction. It binds to receptors on muscle cells, leading to muscle contraction.
ACh esterase, which breaks ACh down into an acetyl group and a choline group. This is then taken up by the neurone and some is reformed into ACh.
acetylcholine (ACh)
Acetylcholine (ACh)
Chemical A would be the best muscle relaxant. Since the muscle relies on ACH as a stimulant, this chemical would inhibit communication between the muscle cell and the neuron thus making it not contract. While Chemical B would cause further contraction of the muscle since calcium is needed to help the myosin head to attach to the actin filliment which creates muscle contraction.
The purpose of the muscle cell is movement, support, help the heart pump and to heart and warmth\ by H.L
Botulin is best described as a neurotoxin that inhibits the release of acetylcholine (ACh) at the neuromuscular junction, leading to muscle paralysis.
None. Muscles only contracts. In order to extend, the muscle on the opposite movement has to contract. For example, the biceps help flex the elbow, but it does not extend the elbow; the triceps does that.
Cholinergic fibers release acetylcholine (ACh) at their synapses to transmit signals to target cells, including muscle cells, gland cells, and other neurons. This neurotransmitter is vital in the functioning of the parasympathetic nervous system and at neuromuscular junctions for muscle contraction.
Acetylcholine is the neurotransmitter at the neuromuscular junction.
Occurs when acetylcholine (ACh) accumulates in the neuromuscular junction. It is called a tetanic contraction.
If enough ACh is released, the sarcolemma at that point becomes temorarily more permeable to sodium ions, which rush into the muscle cell and to potassium ions which diffuse out of the cell. Although more sodium ions enter than posassium ions leave.