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How does facioscapulohumeral muscular syndrome effected the sexual life of men?
Let me first correct you.There is no such thing as facioscapulohumeral muscular syndrome. There is only facioscapulohumeral muscular dystrophy and i don't know if you meant that but if you did then use a little imagination!And really?Asking this type of question??
What is the name of the disease in which a person sleeps with open eyes?
It could be facioscapulohumeral muscular dystrophy.
What has the author Philip Edward Jardine written?
Philip Edward Jardine has written: 'A clinical and molecular study of facioscapulohumeral muscular dystrophy'
When do symptoms of Facioscapulohumeral muscular dystrophy first occur?
most commonly begin in the teens or early twenties, though infant or childhood onset is possible. Symptoms tend to be more severe in those with earlier onset. The disease is named for the regions of the body most severely affected
What are the early symptoms of Facioscapulohumeral muscular dystrophy?
first symptom noticed is often difficulty lifting objects above the shoulders. The weakness may be greater on one side than the other. Shoulder weakness also causes the shoulder blades to jut backward, called scapular winging
What age do you die from facioscapulohumeral muscular dystrophy?
with a lite exercise and good and balance nutrition , the fsh MD patient lives normal life. execess or very less exercise cause muscle damage which may cause future problems. even i am a patient of it too and at age of 23 i am living a good life. if you have any other question feel free to contact me at inder@who.net .
How is Muscular Dystrophy tested for?
In the 1860s, descriptions of boys who grew progressively weaker, lost the ability to walk, and died at an early age became more prominent in medical journals. In the following ten years, French neurologist Guillaume Duchenne gave a comprehensive account of 13 boys with the most common and severe form of the disease (which now carries his name - Duchenne muscular dystrophy). It soon became evident that the disease had more than one form, and that these diseases affected males of all ages. Nine diseases including Duchenne, Becker, limb girdle, congenital, facioscapulohumeral, myotonic, oculopharyngeal, distal, and Emery-Dreifuss are always classified as muscular dystrophy.
Facioscapulohumeral muscular dystrophy?
DefinitionFacioscapulohumeral muscular dystrophy is progressive muscle weakness and loss of muscle tissue.See also: Muscular dystrophyAlternative NamesLandouzy-Dejerine muscular dystrophyCauses, incidence, and risk factorsFacioscapulohumeral muscular dystrophy affects the upper body, unlike Duchenne muscular dystrophy and Becker muscular dystrophy, which affect the lower body.Facioscapulohumeral muscular dystrophy is a genetic disorder. It appears in both men and women and may develop in a child if either parent carries the gene for the disorder.Facioscapulohumeral muscular dystrophy affects approximately 5 out of 100,000 people. It affects men and women equally.SymptomsFacioscapulohumeral muscular dystrophy mainly affects the face, shoulder, and upper arm muscles. However, it can affect muscles around the pelvis, hips, and lower leg.Symptoms often do not appear until age 10 - 26, but it is not uncommon for symptoms to appear much later. In some cases, symptoms never develop.Symptoms are usually mild and very slowly become worse. Facial muscle weakness is common, and may include:Eyelid droopingInability to whistleDecreased facial expressionDepressed or angry facial expressionDifficulty pronouncing wordsShoulder muscle weakness causes deformities such as pronounced shoulder blades (scapular winging) and sloping shoulders. The person has difficulty raising the arms because of shoulder and arm muscle weakness.Weakness of the lower legs is possible as the disorder gets worse. The weakness can be severe enough to interfere with walking.Hearing loss and abnormal heart rhythms may occur, but are rare.Signs and testsA physical examination reveals weakness of the facial and shoulder muscles. High blood pressure may be noted but is usually mild. An eye exam may show changes in the blood vessels in the back of the eye.Tests that may be done include:Creatine kinasetest (may be slightly high)Electrocardiogram (EKG)EMG(electromyography)Genetic testing of chromosome 4Hearing testMuscle biopsy(may confirm the diagnosis)TreatmentThere is no known cure for facioscapulohumeral muscular dystrophy. Treatments are given to control symptoms and improve the person's quality of life. Activity is encouraged. Inactivity such as bedrest can make the muscle disease worse. Physical therapy may help maintain muscle strength.Expectations (prognosis)Disability is often minor. Lifespan is usually not affected.ComplicationsDecreased mobilityDecreased ability to care for selfDeformities of the face and shouldersHearing lossVision loss (rare)Calling your health care providerCall for an appointment with your health care provider if symptoms of this condition develop.Genetic counseling is recommended for couples with a family history of this condition who wish to have children.ReferencesKliegman RM, Behrman RE, Jenson HB, Stanton BF. Muscular dystrophies. In: Kliegman RM, Behrman RE, Jenson HB, Stanton BF, eds. Nelson Textbook of Pediatrics. 18th ed. Philadelphia, Pa: Saunders Elsevier; 2007:chap 608.
What Terminal disease begins with M?
Multiple sclerosis? Very sad that MS is around. Multiple sclerosis (abbreviated MS, also known as disseminated sclerosis or encephalomyelitis disseminata) is an autoimmune condition in which the immune system attacks the central nervous system, leading to demyelination.[1] Disease onset usually occurs in young adults, and it is more common in women.[2] It has a prevalence that ranges between 2 and 150 per 100,000.[3] MS was first described in 1868 by Jean-Martin Charcot.[4] MS affects the areas of the brain and spinal cord known as the white matter, destroying a fatty layer called the myelin sheath, which wraps around nerve fibers and electrically insulates them. When myelin is lost, the axons of neurons can no longer effectively conduct action potentials.[1] The name multiple sclerosis refers to the scars (scleroses - better known as plaques or lesions) in the white matter.[4] Although much is known about the mechanisms involved in the disease process, the cause remains unknown. Theories include genetics or infections. Different environmental risk factors have also been found.[5][1] Almost any neurological symptom can appear with the disease, and often progresses to physical and cognitive disability.[1] MS takes several forms, with new symptoms occurring either in discrete attacks (relapsing forms) or slowly accumulating over time (progressive forms).[6] Between attacks, symptoms may go away completely, but permanent neurological problems often occur, especially as the disease advances.[6] There is no known cure for MS. Treatments attempt to return function after an attack, prevent new attacks, and prevent disability.[1] MS medications can have adverse effects or be poorly tolerated, and many patients pursue alternative treatments, despite the lack of supporting scientific study. The prognosis is difficult to predict, it depends on the subtype of the disease, the individual patient's disease characteristics, the initial symptoms and the degree of disability the person experiences as time advances.[7] Life expectancy of patients is nearly the same as that of the unaffected population.[7] \Or could it be muscular dystrophy you are looking for? Muscular dystrophy (MD) refers to a group of genetic, hereditary muscle diseases that cause progressive muscle weakness.[1][2] Muscular dystrophies are characterized by progressive skeletal muscle weakness, defects in muscle proteins, and the death of muscle cells and tissue.[3] Nine diseases including Duchenne, Becker, limb girdle, congenital, facioscapulohumeral, myotonic, oculopharyngeal, distal, and Emery-Dreifuss are always classified as muscular dystrophy[4] but there are more than 100 diseases in total with similarities to muscular dystrophy. Most types of MD are multi-system disorders with manifestations in body systems including the heart, gastrointestinal and nervous systems, endocrine glands, skin, eyes and other organs.[4]
