well we know during severe hypertension narrowing of arterioles and arteries takes place it is common
because of narrowed arteries provides resistance to blood flow hence its flow to lower extremeties ceases and glomerular filtration doesnot occur properly
as filtration decreases the sodium ions and chloride ions are not filtered and remained in the blood we can note that there is a decreased amount of sodium ions in distal tubule of nephron so there is insufficient of ions to transport across the macula densa cells of juxtraglomerular apparatus which are very close to distal tubule of nephron thus on compensation these cells triggers to release renin, after several pathway in the body like renin cleaves a peptide called angiotensinogen -->angiotensin-1---->angiotensin-2(A-2), so this A-2 is useful for glomerular filtration by constricting efferent arteriole more and more than afferent arteriole in order to increase the amount of blood to remain in glomerulus for filtration,,, people might expect that here the glomerular filtration occurs and more sodium ions are excreted but how long ? no it happens for short time because A-2 is responsible for constriction of important arteries and arterioles mainly happens when hypertension is severe thus blood flow decreases and even sufficent blood doesnt reaches to afferent arteriole of glomerulus thus filtration ceases and sodium and chloride ions are not excreted
beside A-2 is also responsible for formation of aldosterone whose main function is retention of water sodium and chloride ions in distal tubule after filtration but the potassium ions are excreted
silly !!!!!!!this stupid A-2 is also responsible for antidiuretic hormone from pituatary gland thus totally retention retention retention of water sodiium and chloride ;;;;;;he he he
ok so the answer is NO, DURING HYPERTENSION the sodium doesnot excreted in large quantities nevertheless less than the normal
so this is the physiology on the view of this, we can probably say the answer : NO
"of my level best i explained if any thing wrong please reply"
if any thing ambigious please reply
If the reabsorption of sodium by the renal tubule is inhibited, it can lead to increased sodium excretion in the urine, which can result in higher urine output (diuresis). This can lead to loss of water and electrolytes, potentially causing dehydration, electrolyte imbalances, and alterations in blood pressure.
Caffeine: Acts as a diuretic by increasing blood flow to the kidneys. Alcohol: Inhibits the release of vasopressin, a hormone that helps the body retain water. Certain medications: Such as diuretics, used to treat conditions like hypertension and heart failure. High-sodium foods: Can increase urine output by promoting water excretion.
If urine output is greater than intake a pressure differential will exist between inside and outside the urethra and as a result you will urinate your internal organs out.
Urine contain sodium from sodium chloride.
There are many things that affect an animal's urine output such as food intake. Beverage intake also affects an animal's urine output.
For a male OR female, drinking about 2 liters of fluid a day, the normal urine output should be 800 to 2,000 milliliters a day. Urine output below 500 milliliters a day is considered a low urine output and is a sign of disease.
If sodium hydroxide solution and copper tetraoxosulphate were added to urine, a color change might occur. This reaction produces a blue color due to the formation of a complex between copper ions and proteins in urine. This test can be used to detect the presence of proteins in the urine.
Certainly. Decrease cardiac output would mean a decreased in blood flow to the kidneys, which would lead to reduced filtration, therefore urine output.
Decreased urine output is also known as Oliguria. Renal malfunctions or dehydration can cause Oliguria. The ICD-9 code for decreased urine output/oliguria is 788.5.
There are various factors which might decrease urine output. For example, if a person is dehydrated then they will urinate less.
Thiazides like hydrochlorotiazyde is a diuretic and it will increase the volume of urine.
This syndrome occurs when a woman with pre-existing chronic hypertension begins to have protein in the urine after week 20 of pregnancy.